Sadaf Farnam Nia
1 
, Hamid Nasri
1 , Simin Mazaheri Tehrani
2 , Shiva Rouzbahani
3* 1 Nickan Research Institute, Isfahan, Iran
2 Department of Molecular Biotechnology and Health Sciences, School of Medicine, University of Turin, Turin, Italy
3 Bascom Palmer Eye Institute, Miller School of Medicine, University of Miami, Miami, FL, USA
Abstract
Tuberculosis (TB) is a chronic infectious disease caused by Mycobacterium tuberculosis, which primarily affects the lungs but can also involve other organs, including the kidneys. TB has the potential to result in the development of chronic renal failure and requires prompt diagnosis and treatment for effective management. Renal TB can lead to various complications, including renal failure, perinephritis, perinephric abscesses, fistulae, psoas abscesses, and non-functioning kidneys. Additionally, TB-associated nephropathy is an infrequent variant of glomerulonephritis characterized by the deposition of immune complexes containing TB antigens in the kidney’s glomeruli.